Students denoted by *
Jones*, C. , A. Calixto, B. Manes, E. Blair, S. Alvarado, and J.L. Brewster. 2015. Nanoparticle-induced stress eladds to elevated reactive oxygen species, punctate mitochondria, and apoptosis in human lung cells. Southern California Conferences for Undergraduate Research. OS-3-4:00.
Jones*, C.Y., A. Calixto, B. Manes, S.K. Alvarado, J.L. Brewster. 2015. Nanoparticle-induced stress leads to elevated reactive oxygen species, punctate mitochondria, and apoptosis in human lung cells. Southern California Conferences for Undergraduate Research. OS3-B470-4:00.
Manes* B., S. Alvarado*, and J.L. Brewster 2013. Nanoparticle exposure activates an inositol trisphosphate receptor-dependent elevation of reactive oxygen species and apoptosis in human alveolar lung cells, American Society for Cell Biology Annual Meeting (New Orleans, LA), Session: Proteostasis, Cell Stress, and Aging, Abstract 1470.
Mercado* V., and J.L. Brewster. 2013. The surviving and clAP1 anti-apoptotic proteins are differentially down regulated in response to endoplasm reticulum stress. Southern California Conferences for Undergraduate Research. Abstract OS2-20.
Mercado* V., and J.L. Brewster 2013. The Survivin and cIAP1 anti-apoptotic proteins are destabilized during endoplasmic reticulum stress, American Society for Cell Biology Annual Meeting (New Orleans, LA), Session: Proteostasis, Cell Stress, and Aging, Abstract 1472.
Vargas* A., and J.L. Brewster. 2013. Assessment of BHK-21 cell viability at low and high concentrations of Tunicamycin after transfection of Bcl-2 ER localized anti-apoptotic protein. Southern California Conferences for Undergraduate Research. Abstract PSI-9.
Manes* B., Alvarado* S., and J.L. Brewster. 2012. Nanoparticle accumulation in human alveolar cells induce reactive oxygen species and ER stress signaling, American Society for Cell Biology Annual Meeting (San Franciso CA), Session: Proteostasis, Cell Stress, and Aging, Poster
Scholl* A., Osborne* A., Lumley* E., Scott* J., and J.L. Brewster. 2012. Moderate ER stress induces p44/p42 kinase activation and an ITPR-dependent apoptosis, American Society for Cell Biology Annual Meeting (San Franciso CA), Session: Proteostasis, Cell Stress, and Aging, Poster.
Students denoted by *
Murphy* L.A., E.A. Ramirez*, V.T. Trinh*, A.M. Herman*, V.C. Anderson*, and J.L. Brewster 2011. "Endoplasmic reticulum stress or mutation of an EF-hand Ca2+-binding domain directs the FKBP65 rotamase to an ERAD-based proteolysis", Cell Stress and Chaperones, 16:607-619.
Loucks F.A., E.K. Shcroeder, A. Zommer, S. Hilger*, N.A. Kelsey, R.J. Bouchard, C. Blackstone, J.L. Brewster, D.A. Linseman. 2009. Caspases indirectly regulate cleavage of the mitochondrial fusion GTPase OPA1 in neurons undergoing apoptosis, Brain Research, 1250:63-74.
Brewster, J.L., D.A. Linseman, R.J. Bouchard, F.A. Loucks, T.A. Precht, E.A. Esch* and K.A. Heidenreich. 2006. Endoplasmic reticulum stress and tropic factor withdrawal activate distinct signaling cascades that induce glycogen synthase kinase-3b and a caspase-9-dependent apoptosis in cerebellar granule neurons. Molecular and Cellular Neuroscience 32:242-253.
Niederer*, K., D. Morrow*, J. Gettings*, M. Irick*, A. Krawiecki* and J.L. Brewster. 2005. Cypermethrin blocks a mitochondria-dependent apoptotic signal initiated by deficient N-linked glycosylation within the endoplasmic reticulum, Cellular Signalling 17 (2):177-186.
